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Treatment response should be tracked with redox proteomics (cysteine-switch/oxidized-PTP signatures), peroxiredoxin redox states (as sentinels of H 2 O 2 flux) and lipid-peroxide markers (F 2 -isoprostanes, oxidized phospholipids), enabling adaptive dosing that preserves beneficial redox signalling in normal tissues while exploiting malignant redox liabilities (Kisty et al

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